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Rehabilitation Practice and Science

Translated Title

遲發型缺氧性腦病變後之復健：病例報告

Abstract

Carbon monoxide poisoning from accidents or suicide attempts often result in delayed post-hypoxic encephalopathy. Patients with delayed post-hypoxic encephalopathy typically present with recurrent apathy, confusion, agitation, and/or progressive neurological deficits, despite apparent transient recovery. Autopsy reports often reveal diffuse demyelination of subcortical white matter, but the exact pathogenesis is not clear. While comprehensive physical examination and history taking is necessary in diagnosing delayed post-hypoxic encephalopathy, magnetic resonance imaging can be very helpful in the diagnostic work-up. Rehabilitation for severe post-hypoxic encephalopathy can prevent complications such as immobilization and can improve patients' functional status.We report the history, diagnosis, and rehabilitative outcome of a 41-year-old man who developed delayed post-hypoxic encephalopathy one month after carbon monoxide poisoning. Severe neurological deficits were noted in this patient, including bilateral vocal cord paralysis and vegetative status. After about one year of intensive rehabilitation, the patient achieved significant improvements in his functional status and activities of daily living.

Language

Traditional Chinese

First Page

111

Last Page

117

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